Listen folks, myopia, it’s a big deal, very common. We’re talking about eyes here, eyes that are growing too long, believe me. And what happens? Light doesn’t hit where it should. It’s a problem, a huge problem. Now, we’ve got these animal experiments, sure, but they’re not perfect. They’re not the real deal like human myopia. We need the real deal.

So, what’s the best thing we’ve got? Aqueous humor from cataract surgery – it’s fantastic, the best. We’ve looked at the studies, done the research. We searched everywhere, PubMed, you name it, with keywords like “myopia,” “aqueous humor,” “protein.” And we found stuff, important stuff.

The substances in the aqueous humor, they’re different in myopic people. It’s about the scleral matrix breaking down, inflammation, fibrosis, blood vessels, and inhibition. It’s complicated, very complicated. There’s not just one reason for myopia, there are many, and we’re going to find them all.

But here’s the thing, the mechanism of myopia, it’s still a mystery. We don’t have all the answers yet, but we will, we will. So, what do we do? We focus on stopping the scleral matrix from breaking down, we boost the collagen in the sclera, and we fight the inflammation. That’s how we’re going to beat myopia.

And this aqueous humor research, it’s going to give us new insights, the best insights. We’re going to learn so much, and we’re going to use that knowledge to win against myopia. That’s what we’re going to do.